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A Case of Glycogenosis I

Eun SY, Kim SH, Rhee SI, Kim SW, Cho HJ, Park IK

  • KMID: 1698926
  • J Korean Pediatr Soc.
  • 1984 Apr;27(4):401-406.
No abstract available.
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Alterations of Neuropeptides and Neutrotrophic Factors in Kindled Seizures

Eun SY

  • KMID: 1816478
  • J Korean Epilepsy Soc.
  • 2000 Dec;4(2):147-149.
No abstract available.
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A Case of Renal Tuberculosis in a Child

Cha MY, Eun SY, Lee CG, Lee SI, Kim SW

  • KMID: 1698883
  • J Korean Pediatr Soc.
  • 1984 Jul;27(7):733-737.
No abstract available.
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Crohn's Disease with Oral and Tonsillar Ulceration

Eun SY, Lee SH, Park SJ, Kim HJ

  • KMID: 2274715
  • Korean J Otolaryngol-Head Neck Surg.
  • 2000 May;43(5):555-558.
Granulomatous lesions of the small bowel and colon are common in Crohn's disease, with other sites of involvement relatively infrequent. Nevertheless, Crohn's disease is frequently described as a disease of...
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Long-term Synaptic Plasticity: Circuit Perturbation and Stabilization

Park JM, Jung SC, Eun SY

At central synapses, activity-dependent synaptic plasticity has a crucial role in information processing, storage, learning, and memory under both physiological and pathological conditions. One widely accepted model of learning mechanism...
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Sustained K+ Outward Currents are Sensitive to Intracellular Heteropodatoxin2 in CA1 Neurons of Organotypic Cultured Hippocampi of Rats

Jung SC, Eun SY

Blocking or regulating K+ channels is important for investigating neuronal functions in mammalian brains, because voltage-dependent K+ channels (Kv channels) play roles to regulate membrane excitabilities for synaptic and somatic...
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Expression of Fibroblast Growth Factor Receptor 3 in the Recurrence of Non-Muscle-Invasive Urothelial Carcinoma of the Bladder

Maeng YH, Eun SY, Huh JS

  • KMID: 2314806
  • Korean J Urol.
  • 2010 Feb;51(2):94-100.
PURPOSE: The fibroblast growth factor receptor 3 (FGFR3) gene is known to be frequently mutated in noninvasive urothelial carcinomas of the bladder. In this study, we investigated the expression of...
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Treatment of Adult-Onset Otitis Media with Effusion by Morphologic Difference of Nasopharynx and Eustachian Tube Orifice

Yoon YJ, Jeong JY, Lee DS, Eun SY, Lee SH, Lee SW, Lee SC

  • KMID: 1559627
  • Korean J Otolaryngol-Head Neck Surg.
  • 1998 May;41(5):578-581.
BACKGROUND AND OBJECTIVES: Recently, adult-onset otitis media with effusion(OME) tends to increase in its incidence. We compared the treatment of OME by morphologic difference of nasopharynx and eustachian tube orifice...
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DAMGO, a micro-opioid agonist and cholecystokinin-octapeptide have dual modulatory effects on capsaicin-activated current in rat dorsal root ganglion neurons

Eun SY, Kim J, Lee J, Jung SJ, Park JM, Park YK, Kim D, Kim SJ, Kwak J, Kim J

  • KMID: 1606841
  • Korean J Physiol Pharmacol.
  • 2001 Feb;5(1):71-78.
Capsaicin, a pungent ingredient of hot pepper, elicits an intense burning pain when applied cutaneously and intradermally. Activation of capsaicin-gated channel in. C-type dorsal root ganglion (DRG) neurons produces nonselective cationic currents. Although...
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Responses of dorsal horn neurons to peripheral chemical stimulation in the spinal cord of anesthetized cats

Jung SJ, Park JM, Lee JH, Lee JH, Eun SY, Kim SJ, Lim WI, Cho SH, Kim J

  • KMID: 1580631
  • Korean J Physiol Pharmacol.
  • 2000 Feb;4(1):15-24.
Although nociceptive informations are thought to be processed via different neural mechanisms depending on the types of stimuli, sufficient data have not been accumulated yet. We performed a series of experiments to elucidate...
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The Downregulation of Somatic A-Type K+ Channels Requires the Activation of Synaptic NMDA Receptors in Young Hippocampal Neurons of Rats

Kang MS, Yang YS, Kim SH, Park JM, Eun SY, Jung SC

The downregulation of A-type K+ channels (IA channels) accompanying enhanced somatic excitability can mediate epileptogenic conditions in mammalian central nervous system. As IA channels are dominantly targeted by dendritic and...
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Chronic Ca²⁺ influx through voltage-dependent Ca²⁺ channels enhance delayed rectifier K⁺ currents via activating Src family tyrosine kinase in rat hippocampal neurons

Yang YS, Jeon SC, Kim DK, Eun SY, Jung SC

Excessive influx and the subsequent rapid cytosolic elevation of Ca²⁺ in neurons is the major cause to induce hyperexcitability and irreversible cell damage although it is an essential ion for...
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Neuroprotective mechanisms of dieckol against glutamate toxicity through reactive oxygen species scavenging and nuclear factor-like 2/heme oxygenase-1 pathway

Cui Y, Amarsanaa , Lee JH, Rhim JK, Kwon JM, Kim SH, Park JM, Jung SC, Eun SY

Glutamate toxicity-mediated mitochondrial dysfunction and neuronal cell death are involved in the pathogenesis of several neurodegenerative diseases as well as acute brain ischemia/stroke. In this study, we investigated the neuroprotective...
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Nobiletin attenuates neurotoxic mitochondrial calcium overload through K⁺ influx and ΔΨ(m) across mitochondrial inner membrane

Lee JH, Amarsanaa , Wu J, Jeon SC, Cui Y, Jung SC, Park DB, Kim SJ, Han SH, Kim HW, Rhyu IJ, Eun SY

Mitochondrial calcium overload is a crucial event in determining the fate of neuronal cell survival and death, implicated in pathogenesis of neurodegenerative diseases. One of the driving forces of calcium...
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Cell type-specific upregulation of myristoylated alanine-rich C kinase substrate and protein kinase C-alpha, -beta I, -beta II, and -delta in microglia following kainic acid-induced seizures

Eun SY, Kim EH, Kang KS, Kim HJ, Jo SA, Kim SJ, Jo SH, Kim SJ, Blackshear PJ, Kim J

  • KMID: 1094157
  • Exp Mol Med.
  • 2006 Jun;38(3):310-319.
Myristoylated alanine-rich C kinase substrate (MARCKS) is a widely distributed protein kinase C (PKC) substrate and has been implicated in actin cytoskeletal rearrangement in response to extracellular stimuli. Although MARCKS...
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Dieckol Attenuates Microglia-mediated Neuronal Cell Death via ERK, Akt and NADPH Oxidase-mediated Pathways

Cui Y, Park JY, Wu J, Lee JH, Yang YS, Kang MS, Jung SC, Park JM, Yoo ES, Kim SH, Ahn Jo S, Suk K, Eun SY

Excessive microglial activation and subsequent neuroinflammation lead to synaptic loss and dysfunction as well as neuronal cell death, which are involved in the pathogenesis and progression of several neurodegenerative diseases....
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