- Molecular Mechanism of Reactive Oxygen Species-dependent ASK1 Activation in Innate Immunity
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Yamauchi S, Noguchi T, Ichijo H
- KMID: 2054252
- Immune Netw.
- 2008 Mar;8(1):1-6.
- doi: 10.4110/in.2008.8.1.1
Apoptosis signal-regulating kinase 1 (ASK1), a mitogen- activated protein kinase kinase kinase, plays pivotal roles in stress responses. In addition, ASK1 has emerged as a key regulator of immune responses... -
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- Generation and Characterization of Monoclonal Antibodies against Human Interferon-lambda1
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Hong SH, Kim JS, Park S
- KMID: 2054253
- Immune Netw.
- 2008 Mar;8(1):7-12.
- doi: 10.4110/in.2008.8.1.7
BACKGROUND: Members belonging to the interferon-lambda (IFN-lambda) family exert protective action against viral infection; however, the mechanisms of their action have remained elusive. To study IFN-lambda biology, such as endocytosis... -
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- IL-23 P19 Expression Induced by IL-17 and IL-1beta in Rheumatoid Arthritis Synovial Mononuclear Cells
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Cho ML, Heo YJ, Oh HJ, Kang CM, Lee SY, Hong YS, Kim HY
- KMID: 2054256
- Immune Netw.
- 2008 Mar;8(1):29-37.
- doi: 10.4110/in.2008.8.1.29
Interleukin-23 (IL-23) is a novel pro-inflammatory cytokine which has been implicated to play a pathogenic role in rheumatoid arthritis (RA). This study was undertaken to investigate the IL-23 inductive activity... -
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- Coptis chinensis Extract Inhibits the Production of Inflammatory Mediators and Delayed Type Hypersensitivity in Mice
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Lee YA, Hong SJ, Lee SH, Kim KS, Park EK, Jung K, Han CS, Yoo MC, Yang HI
- KMID: 2054254
- Immune Netw.
- 2008 Mar;8(1):13-20.
- doi: 10.4110/in.2008.8.1.13
BACKGROUND: Coptis chinensis rhizome has been used as a medicinal herb in traditional Oriental medicine. We investigated the effects of Coptis chinensis extract on inflammatory mediators and delayed type hypersensitivity... -
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- A Co-inhibitory Molecule, B7-H4, Synergistically Potentiates Oral Tolerance by Inducing CD4+CD25+FoxP3+ T Cells
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Wen L, Yang S, Choi J, Kim Y, Kwon E, Lee H, Jeoung H, Hwang D, Hwang D, Choi I
- KMID: 2054255
- Immune Netw.
- 2008 Mar;8(1):21-28.
- doi: 10.4110/in.2008.8.1.21
BACKGROUND: A co-inhibitory molecule, B7-H4, is believed to negatively regulate T cell immunity by suppressing T cell proliferation and inhibiting cytokine production. However, the mechanism behind B7-H4-mediated tolerance remains unclear. METHODS:... -
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