J Cardiovasc Ultrasound.  2009 Jun;17(2):73-75. 10.4250/jcu.2009.17.2.73.

A Case of Myocardial Abscess Mimicking Acute Myocardial Infarction

Affiliations
  • 1Department of Cardiology, The Heart Center, Chonnam National University Hospital, Gwangju, Korea. jcpark54@hanmail.net

Abstract

Myocardial abscess, a rare and life-threatening disease, occurs as a complication of infective endocarditis, acute myocardial infarction, or other infections in the setting of debilitating condition. We report a case of myocardial abscess mimicking acute myocardial infarction (AMI), not a following consequence of AMI. We can diagnose the disease with the aid of transthoracic echocardiography and cardiac computed tomography.

Keyword

Abscess; Myocardial infarction

MeSH Terms

Abscess
Echocardiography
Endocarditis
Myocardial Infarction

Figure

  • Fig. 1 Electrocardiogram on admission (A) shows ST segment elevations and T wave inversions in the anterior leads. Follow-up electrocardiogram checked 7 weeks later after the first exam (B) reveals normalization of ST-segment abnormality.

  • Fig. 2 Apical 4 chamber view (A) and apical 2 chamber view (B) of transthoracic echocardiography on admission shows hypoechoic lesion (arrow) at the myocardium just adjacent hypokinetic apical area. Note that the maximal size was 27.2×11.9 mm.

  • Fig. 3 Volume rendering image (A) of cardiac computed tomography shows no critical narrowing in either coronary arteries. Cardiac computed tomography (B, C and D) demonstrates mild diffuse thickening with contrast enhancement of apical myopericardium and loculated fluid collection in the area (arrows). Note that left anterior descending coronary artery runs beside the lesion (arrowhead in panel B).

  • Fig. 4 2D Echocardiogram performed 2 weeks later (A and B) shows no significant interval change of lesion size. 2D echocardiogram carried out 7 weeks later (C and D) demonstrates complete resolution of the lesion.

  • Fig. 5 Follow-up chest computed tomography after 3 weeks of antibiotics treatment shows markedly decreased lesion, but still remained focal wall thickening and enhancement.


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