- Two Case of Primary Aldosteronism Induced by Aldosterone Producing Adrenal Adenoma in a Family
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Jang YR, Kim SH, Eom YS, Lee KY
- KMID: 1973799
- Endocrinol Metab.
- 2012 Dec;27(4):329-333.
- doi: 10.3803/EnM.2012.27.4.329
Primary aldosteronism, is defined as a group of disorders characterized by the excess of aldosteron, with suppressed rennin activity, resulting in hypertension and hypokalemia. In most cases, primary aldosteronism is... -
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- A Case of Licorice Induced Hypokalemic Paralysis with Rhabdomyolysis
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Hong HI, Han KH, Hwang JW, Kim YD, Shim MS, Kim JY
- KMID: 2331200
- J Korean Soc Endocrinol.
- 2005 Apr;20(2):179-182.
Prolonged ingestion of licorice can cause hypermineralocorticoidism, with sodium retention, potassium loss and hypertension. Nevertheless, its initial presentation with a very severe degree of hypokalemic paralysis and rhabdomyolysis are exceedingly... -
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- ANCA-related crescentic glomerulonephritis in a patient with systemic sclerosis
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Kwon JS, Shin YK, Kwak JH, Park SH, Kim SK, Choe JY
- KMID: 2081675
- Korean J Med.
- 2009 Apr;76(Suppl 1):S154-S157.
The renal manifestations of systemic sclerosis include proteinuria, hypertension, azotemia, and renal crisis. Two types of scleroderma renal crisis (SRC) are recognized. Typical SRC is a syndrome consisting of acute-onset... -
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- A Case of Male Pseudohermaphroditism due to 17alpha-Hydroxylase Deficiency
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Park KA, Chung YK, Lee JR, Choi YM, Lee GH, Kim HS, Jee BC, Ku SY, Suh CS, Kim SH, Kim JG, Moon SY, Kim SY
- KMID: 1830752
- Korean J Fertil Steril.
- 2006 Jun;33(2):133-138.
Female phenotype of a 46,XY male may originates from male pseudohermaphroditism due to 17alpha-hydroxylase deficiency. Lack of cortisol increases adrenocorticotropic hormone (ACTH) and mineralocorticoid production, leading to low renin hypertention... -
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- 1 Case of Primary Aldosteronism Caused by Adrenal Hyperplasia
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Park SH, Choi SB, Kim CS
- KMID: 2373897
- Yeungnam Univ J Med.
- 1986 Dec;3(1):313-318.
- doi: 10.12701/yujm.1986.3.1.313
Primary aldosteronism is a disease that the stimulus for the excessive aldosterone production residues within the adrenal gland. It was first described by conn in 1955. And many cases were reported... -
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